Bifalgorithm post
38 Ma complaining of che pen since four hours. What is this? Intituagnosis calertri and treatment? EC shows inferior lead stvision. Diagnosis is inferior vol miarddial infection. Calpriated artery is right coronary artryy treatment is loading those throvolics using Atq, rapa and angels that is pretty today. Drug of discussion is steppto. Mechanism. Action. P plasminogen is converted to P plasmid and then fibrin is converted to fibril fragment. Sdk activates a plasminogen conversion to plasmid so fibriine degradation occur. Special points obtained fromtaollyticto occur. Group C. It is an important MCQ. It is considered only when the cost, tissue or ability of tpa. It is non-fibrin specific so depleted circulating fibrinogen and period. Process DE bleeding deep after 30 to 80 minutes goes for MI 7.5 to 15 like international unit IV over 60 minutes for DEV pulmonary eolism 2.5 LA IU loading do over one hour, then one LA international unit for 24 hour. Side breeding. Hy pottension, n. Alexis resistance, thank you stay tuned.
38 Ma complaining of che pen since four hours. What is this? Intituagnosis calertri and treatment? EC shows inferior lead stvision. Diagnosis is inferior vol m…
Ideas in this flowchart
- 38 Ma complaining of chest pain
- Diagnosis and Treatment
- Inferior Myocardial Infarction
- Right Coronary Artery
- Thrombolytics Treatment
- Drugs: Atq, Rapa, Angels
- Steptokinase
- Mechanism of Action
- Plasminogen to Plasmin Conversion
- Fibrin to Fibrin Fragments
- Sdk Activation
- Special Points
- Group C MCQ Importance
- Non-fibrin Specific
- Depleted Circulating Fibrinogen
- Bleeding Process
- Deep Bleeding after 30-80 Minutes
- Dosage
- MI: 7.5-15 IU IV over 60 Minutes
- Pulmonary Embolism: 2.5 LA IU Loading
- Side Effects
- Hypotension
- Allergic Reactions